Molecular programs induced by heat acclimation confer neuroprotection against TBI and hypoxic insults via cross-tolerance mechanisms

Molecular programs induced by heat acclimation confer neuroprotection against TBI and hypoxic insults via cross-tolerance mechanisms

Neuroprotection following prolonged exposure to high ambient temperatures (heat acclimation HA) develops via altered molecular programs such as cross-tolerance (Heat Acclimation -Neuroprotection Cross-Tolerance -HANCT). The mechanisms underlying cross-tolerance depend on enhanced on-demand protecti...

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Bibliographic Details
Main Authors: Michal eHorowitz, Gali eUmschweif, Assaf eYacobi, Esther eShohami
Format: Article
Language:English
Published: Frontiers Media S.A. 2015-07-01
Series:Frontiers in Neuroscience
Subjects:
Traumatic brain injury and Neuroprotection
Heat acclimation and Cross tolerance
Angiotensin AT2 receptor and neurogenesis
AKT-HIF-1 signaling
Hypoxia - NMDA and AMPA receptors.
Online Access:http://journal.frontiersin.org/Journal/10.3389/fnins.2015.00256/full

Internet

http://journal.frontiersin.org/Journal/10.3389/fnins.2015.00256/full

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