Saluisn-β contributes to endothelial dysfunction in monocrotaline-induced pulmonary arterial hypertensive rats

Saluisn-β contributes to endothelial dysfunction in monocrotaline-induced pulmonary arterial hypertensive rats

Background: The endothelial dysfunction and the consequent attenuated pulmonary vasodilatation are the major causes of elevated pulmonary arterial resistance and pressure in pulmonary arterial hypertension (PAH). Current study aimed to explore the effects of a TOR2A gene product, salusin-β, on endot...

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Bibliographic Details
Main Authors: Yan Pan, Aidong Chen, Xingxing Wang, Changlei Bao, Shuxin Liang, Haiyang Tang, Ying Han
Format: Article
Language:English
Published: Elsevier 2022-11-01
Series:Biomedicine & Pharmacotherapy
Subjects:
Endothelial dysfunction
Endothelium-dependent vasodilatation
TOR2A gene product
Nitric oxide
Reactive oxygen species
Pulmonary arterial hypertension
Online Access:http://www.sciencedirect.com/science/article/pii/S0753332222011374

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http://www.sciencedirect.com/science/article/pii/S0753332222011374

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