MS‐275 potentiates the effect of YM‐155 in lung adenocarcinoma via survivin downregulation induced by miR‐138 and miR‐195

Background YM‐155 has been proven to be an efficient antitumor suppressor in non‐small cell lung cancer (NSCLC) cells. However, the suppressive effect of YM‐155 on the expression of survivin is not sufficient and has a short half‐life. MS‐275, a histone deacetylase inhibitor, has significant antitum...

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Bibliographic Details
Main Authors: Bai‐Ling Luo, Yan Zhou, Hui Lv, Sheng‐Hua Sun, Wen‐Xiang Tang
Format: Article
Language:English
Published: Wiley 2019-06-01
Series:Thoracic Cancer
Subjects:
Online Access:https://doi.org/10.1111/1759-7714.13076