Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease

Excess free iron is a substrate for the formation of reactive oxygen species (ROS), thereby augmenting oxidative stress. Oxidative stress is a well-established cause of organ damage in the liver, the main site of iron storage. Ferroptosis, an iron-dependent mechanism of regulated cell death, has rec...

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Main Authors: Sophie Gensluckner, Bernhard Wernly, Christian Datz, Elmar Aigner
Format: Article
Language:English
Published: MDPI AG 2024-02-01
Series:Antioxidants
Subjects:
Online Access:https://www.mdpi.com/2076-3921/13/2/208
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author Sophie Gensluckner
Bernhard Wernly
Christian Datz
Elmar Aigner
author_facet Sophie Gensluckner
Bernhard Wernly
Christian Datz
Elmar Aigner
author_sort Sophie Gensluckner
collection DOAJ
description Excess free iron is a substrate for the formation of reactive oxygen species (ROS), thereby augmenting oxidative stress. Oxidative stress is a well-established cause of organ damage in the liver, the main site of iron storage. Ferroptosis, an iron-dependent mechanism of regulated cell death, has recently been gaining attention in the development of organ damage and the progression of liver disease. We therefore summarize the main mechanisms of iron metabolism, its close connection to oxidative stress and ferroptosis, and its particular relevance to disease mechanisms in metabolic-dysfunction-associated fatty liver disease and potential targets for therapy from a clinical perspective.
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spelling doaj.art-1bc0cc5f63c24be6a4704c15354a89bf2024-02-23T15:05:30ZengMDPI AGAntioxidants2076-39212024-02-0113220810.3390/antiox13020208Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver DiseaseSophie Gensluckner0Bernhard Wernly1Christian Datz2Elmar Aigner3Department of Internal Medicine I, Paracelsus Medical University, Müllner Hauptstrasse 48, 5020 Salzburg, AustriaDepartment of Medicine, General Hospital Oberndorf, Teaching Hospital of the Paracelsus Medical University, 5110 Oberndorf, AustriaDepartment of Medicine, General Hospital Oberndorf, Teaching Hospital of the Paracelsus Medical University, 5110 Oberndorf, AustriaDepartment of Internal Medicine I, Paracelsus Medical University, Müllner Hauptstrasse 48, 5020 Salzburg, AustriaExcess free iron is a substrate for the formation of reactive oxygen species (ROS), thereby augmenting oxidative stress. Oxidative stress is a well-established cause of organ damage in the liver, the main site of iron storage. Ferroptosis, an iron-dependent mechanism of regulated cell death, has recently been gaining attention in the development of organ damage and the progression of liver disease. We therefore summarize the main mechanisms of iron metabolism, its close connection to oxidative stress and ferroptosis, and its particular relevance to disease mechanisms in metabolic-dysfunction-associated fatty liver disease and potential targets for therapy from a clinical perspective.https://www.mdpi.com/2076-3921/13/2/208iron metabolismoxidative stressferroptosisMASLDliver fibrosis
spellingShingle Sophie Gensluckner
Bernhard Wernly
Christian Datz
Elmar Aigner
Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
Antioxidants
iron metabolism
oxidative stress
ferroptosis
MASLD
liver fibrosis
title Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
title_full Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
title_fullStr Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
title_full_unstemmed Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
title_short Iron, Oxidative Stress, and Metabolic Dysfunction—Associated Steatotic Liver Disease
title_sort iron oxidative stress and metabolic dysfunction associated steatotic liver disease
topic iron metabolism
oxidative stress
ferroptosis
MASLD
liver fibrosis
url https://www.mdpi.com/2076-3921/13/2/208
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