LncRNA H19 inhibition impairs endoplasmic reticulum-mitochondria contact in hepatic cells and augments gluconeogenesis by increasing VDAC1 levels

LncRNA H19 inhibition impairs endoplasmic reticulum-mitochondria contact in hepatic cells and augments gluconeogenesis by increasing VDAC1 levels

Inspite of exerting independent cellular functions, the endoplasmic-reticulum (ER) and the mitochondria also physically connect at specific sites termed mitochondria-associated ER membranes (MAMs) and these sites consist of several tethering proteins that play varied roles in diverse cellular proces...

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Bibliographic Details
Main Authors: Arun Nandwani, Shalu Rathore, Malabika Datta
Format: Article
Language:English
Published: Elsevier 2024-02-01
Series:Redox Biology
Subjects:
lncRNA
Diabetes
Liver
ROS
Gluconeogenesis
JNK
Online Access:http://www.sciencedirect.com/science/article/pii/S2213231723003907

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http://www.sciencedirect.com/science/article/pii/S2213231723003907

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