Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78

Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78

Cardiomyocyte apoptosis is an important cause of trauma-induced secondary cardiac injury (TISCI), in which the endoplasmic reticulum stress (ERS)-mediated apoptosis signaling pathway is known to be first activated, but the mechanism remains unclear. In this study, rat models of traumatic injury are...

Полное описание

Библиографические подробности
Главные авторы: Yan Zi, Liu Yufeng, Yang Bowen, Zhao Wenhui, Wang Yan, Wang Deping, Li Jianguo, Jiao Xiangying, Cao Jimin
Формат: Статья
Язык:English
Опубликовано: China Science Publishing & Media Ltd. 2023-12-01
Серии:Acta Biochimica et Biophysica Sinica
Предметы:
acetylation
apoptosis
cardiomyocytes
GRP78
trauma
Online-ссылка:https://www.sciengine.com/doi/10.3724/abbs.2023277

Internet

https://www.sciengine.com/doi/10.3724/abbs.2023277

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