Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78

Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78

Cardiomyocyte apoptosis is an important cause of trauma-induced secondary cardiac injury (TISCI), in which the endoplasmic reticulum stress (ERS)-mediated apoptosis signaling pathway is known to be first activated, but the mechanism remains unclear. In this study, rat models of traumatic injury are...

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Détails bibliographiques
Auteurs principaux: Yan Zi, Liu Yufeng, Yang Bowen, Zhao Wenhui, Wang Yan, Wang Deping, Li Jianguo, Jiao Xiangying, Cao Jimin
Format: Article
Langue:English
Publié: China Science Publishing & Media Ltd. 2023-12-01
Collection:Acta Biochimica et Biophysica Sinica
Sujets:
acetylation
apoptosis
cardiomyocytes
GRP78
trauma
Accès en ligne:https://www.sciengine.com/doi/10.3724/abbs.2023277

Internet

https://www.sciengine.com/doi/10.3724/abbs.2023277

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