Platelet glycogen synthase kinase 3β regulates plasma β amyloid and phosphorylated tau levels in chronic kidney disease patients with cognitive dysfunction; therapeutic role of erythropoietin

Platelet glycogen synthase kinase 3β regulates plasma β amyloid and phosphorylated tau levels in chronic kidney disease patients with cognitive dysfunction; therapeutic role of erythropoietin

Introduction: Patients with chronic kidney disease (CKD) have increasingly been diagnosed with cognitive impairment. Glycogen synthase kinase 3β (GSK3β) is directly causing both phosphorylated tau (pTau) and amyloid β (Aβ) accumulation in Alzheimer’s disease (AD). GSK3β expression is more abundant i...

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Bibliographic Details
Main Authors: Vinothkumar Ganesan, Krishnakumar Sethuraman, Sureshkumar Sureshkumar, Venkataraman Prabhu
Format: Article
Language:English
Published: Society of Diabetic Nephropathy Prevention 2023-09-01
Series:Journal of Nephropathology
Subjects:
chronic kidney disease
cognitive impairment
aβ
tau protein
gsk3β
recombinant human erythropoietin
Online Access:https://nephropathol.com/PDF/jnp-12-e17238.pdf

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https://nephropathol.com/PDF/jnp-12-e17238.pdf

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