MiR-203-3p inhibits the oxidative stress, inflammatory responses and apoptosis of mice podocytes induced by high glucose through regulating Sema3A expression

MiR-203-3p inhibits the oxidative stress, inflammatory responses and apoptosis of mice podocytes induced by high glucose through regulating Sema3A expression

Diabetic nephropathy (DN) is the most serious long-term microvascular complication of diabetes, which mainly causes podocyte injury. Many studies have shown that microRNAs play a vital role in the development of DN. Studies have shown that miR-203-3p is involved in mesangial cell proliferation and a...

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Bibliographic Details
Main Authors: Chen Jingfu, Xu Qing, Zhang Wei, Zhen YuLan, Cheng Fei, Hua Guo, Lan Jun, Tu Chang
Format: Article
Language:English
Published: De Gruyter 2020-12-01
Series:Open Life Sciences
Subjects:
diabetic nephropathy
mir-203-3p
sema3a
oxidative stress
inflammatory responses
apoptosis
Online Access:https://doi.org/10.1515/biol-2020-0088

Internet

https://doi.org/10.1515/biol-2020-0088

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