Ambient ozone, and urban PM2.5 co-exposure, aggravate allergic asthma via transient receptor potential vanilloid 1-mediated neurogenic inflammation

Ambient ozone, and urban PM2.5 co-exposure, aggravate allergic asthma via transient receptor potential vanilloid 1-mediated neurogenic inflammation

Allergic asthma is the most common pulmonary inflammatory disease, and epidemiological studies have revealed that PM2.5 or ambient ozone (O3) exposure contribute to the higher prevalence of allergic asthma. Current experimental evidence focus principally on the pathogenic effect of exposure to a sin...

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Bibliographic Details
Main Authors: Zongpei Lian, Haomin Qi, Xudong Liu, Yuchao Zhang, Ruijia Xu, Xu Yang, Yan Zeng, Jinquan Li
Format: Article
Language:English
Published: Elsevier 2022-09-01
Series:Ecotoxicology and Environmental Safety
Subjects:
Allergic asthma
PM2.5
Ozone
TRPV1
Substance P
Online Access:http://www.sciencedirect.com/science/article/pii/S0147651322008405

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http://www.sciencedirect.com/science/article/pii/S0147651322008405

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