Vascular injury associated with ethanol intake is driven by AT1 receptor and mitochondrial dysfunction

Vascular injury associated with ethanol intake is driven by AT1 receptor and mitochondrial dysfunction

Background: Renin-angiotensin (Ang II)-aldosterone system (RAAS) is crucial for the cardiovascular risk associated with excessive ethanol consumption. Disturbs in mitochondria have been implicated in multiple cardiovascular diseases. However, if mitochondria dysfunction contributes to ethanol-induce...

Full description

Bibliographic Details
Main Authors: Wanessa M.C. Awata, Juliano V. Alves, Rafael M. Costa, Ariane Bruder-Nascimento, Shubhnita Singh, Gabriela S. Barbosa, Carlos Renato Tirapelli, Thiago Bruder-Nascimento
Format: Article
Language:English
Published: Elsevier 2023-12-01
Series:Biomedicine & Pharmacotherapy
Subjects:
Ethanol
Vascular dysfunction
Mitochondria
Reactive oxygen species
Online Access:http://www.sciencedirect.com/science/article/pii/S0753332223016438

Internet

http://www.sciencedirect.com/science/article/pii/S0753332223016438

Similar Items