Elevated liver glycogenolysis mediates higher blood glucose during acute exercise in Barth syndrome

Elevated liver glycogenolysis mediates higher blood glucose during acute exercise in Barth syndrome

Barth syndrome (BTHS) is an X-linked recessive genetic disorder due to mutations in the Tafazzin (TAFAZZIN) gene that lead to cardiac and skeletal muscle mitochondrial dysfunction. Previous studies in humans with BTHS demonstrate that the defects in muscle mitochondrial oxidative metabolism result i...

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Bibliographic Details
Main Authors: George G. Schweitzer, Grace L. Ditzenberger, Curtis C. Hughey, Brian N. Finck, Michael R. Martino, Christina A. Pacak, Barry J. Byrne, William Todd Cade
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2023-01-01
Series:PLoS ONE
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10470866/?tool=EBI

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10470866/?tool=EBI

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