Intestinal Acyl-CoA synthetase 5 (ACSL5) deficiency potentiates postprandial GLP-1 & PYY secretion, reduces food intake, and protects against diet-induced obesity

Intestinal Acyl-CoA synthetase 5 (ACSL5) deficiency potentiates postprandial GLP-1 & PYY secretion, reduces food intake, and protects against diet-induced obesity

Objective: In the small intestine, the products of digestion of dietary triacylglycerol (TAG), fatty acids (FA) and monoacylglycerol, are taken up by absorptive cells, enterocytes, for systemic energy delivery. These digestion products can also bind receptors on endocrine cells to stimulate the rele...

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Bibliographic Details
Main Authors: John D. Griffin, Ying Zhu, Andrew Reeves, Kimberly K. Buhman, Andrew S. Greenberg
Format: Article
Language:English
Published: Elsevier 2024-05-01
Series:Molecular Metabolism
Subjects:
Intestine
Acyl-CoA long chain synthetase 5
Glucagon like peptide-1
Food intake
Dietary fat absorption
Online Access:http://www.sciencedirect.com/science/article/pii/S2212877824000498

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http://www.sciencedirect.com/science/article/pii/S2212877824000498

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