Neuroprotective effects of ZL006 in Aβ1–42-treated neuronal cells

Neuroprotective effects of ZL006 in Aβ1–42-treated neuronal cells

Amyloid beta (Aβ)-induced neurotoxicity and oxidative stress plays an important role in the pathogenesis of Alzheimer’s disease (AD). ZL006 is shown to reduce over-produced nitric oxide and oxidative stress in ischemic stroke by interrupting the interaction of neuronal nitric oxide synthase and post...

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Bibliographic Details
Main Authors: Wen-Yuan Tao, Lin-Jie Yu, Su Jiang, Xiang Cao, Jian Chen, Xin-Yu Bao, Fei Li, Yun Xu, Xiao-Lei Zhu
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2020-01-01
Series:Neural Regeneration Research
Subjects:
akt; alzheimer’s disease; amyloid-beta; apoptosis; heme oxygenase-1; neurotoxicity; nrf2; oxidative stress; treatment; zl006
Online Access:http://www.nrronline.org/article.asp?issn=1673-5374;year=2020;volume=15;issue=12;spage=2296;epage=2305;aulast=Tao

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http://www.nrronline.org/article.asp?issn=1673-5374;year=2020;volume=15;issue=12;spage=2296;epage=2305;aulast=Tao

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