Increased EID1 nuclear translocation impairs synaptic plasticity and memory function associated with pathogenesis of Alzheimer's disease

Increased EID1 nuclear translocation impairs synaptic plasticity and memory function associated with pathogenesis of Alzheimer's disease

Though loss of function in CBP/p300, a family of CREB-binding proteins, has been causally associated with a variety of human neurological disorders, such as Rubinstein–Taybi syndrome, Huntington's disease and drug addiction, the role of EP300 interacting inhibitor of differentiation 1 (EID1), a...

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Bibliographic Details
Main Authors: Rugao Liu, Joy X. Lei, Chun Luo, Xun Lan, Liying Chi, Panyue Deng, Saobo Lei, Othman Ghribi, Qing Yan Liu
Format: Article
Language:English
Published: Elsevier 2012-03-01
Series:Neurobiology of Disease
Subjects:
EP300 interacting inhibitor of differentiation 1
CBP/p300
Histone
p53
Acetylation
Alzheimer's disease
Online Access:http://www.sciencedirect.com/science/article/pii/S0969996111003822

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http://www.sciencedirect.com/science/article/pii/S0969996111003822

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