Ca2+/calmodulin‐dependent protein kinase II and protein kinase G oxidation contributes to impaired sarcomeric proteins in hypertrophy model

Ca2+/calmodulin‐dependent protein kinase II and protein kinase G oxidation contributes to impaired sarcomeric proteins in hypertrophy model

Abstract Aims Volume overload (VO) induced hypertrophy is one of the hallmarks to the development of heart diseases. Understanding the compensatory mechanisms involved in this process might help preventing the disease progression. Methods and results Therefore, the present study used 2 months old Wi...

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Bibliographic Details
Main Authors: Kamilla Gömöri, Melissa Herwig, Heidi Budde, Roua Hassoun, Nusratul Mostafi, Saltanat Zhazykbayeva, Marcel Sieme, Suvasini Modi, Tamara Szabados, Judit Pipis, Nikolett Farkas‐Morvay, István Leprán, Gergely Ágoston, István Baczkó, Árpád Kovács, Andreas Mügge, Péter Ferdinandy, Anikó Görbe, Péter Bencsik, Nazha Hamdani
Format: Article
Language:English
Published: Wiley 2022-08-01
Series:ESC Heart Failure
Subjects:
Volume‐overload
Hypertrophy
Oxidative stress
Sarcomeric proteins
Online Access:https://doi.org/10.1002/ehf2.13973

Internet

https://doi.org/10.1002/ehf2.13973

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