Increased AR expression in castration-resistant prostate cancer rapidly induces AR signaling reprogramming with the collaboration of EZH2

Increased AR expression in castration-resistant prostate cancer rapidly induces AR signaling reprogramming with the collaboration of EZH2

Elevated androgen receptor (AR) expression is a hallmark of castration-resistant prostate cancer (CRPC) and contributes to the restoration of AR signaling under the conditions of androgen deprivation. However, whether overexpressed AR alone with the stimulation of castrate levels of androgens can be...

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Bibliográfalaš dieđut
Váldodahkkit: Maryam Labaf, Muqing Li, Lily Ting, Breelyn Karno, Songqi Zhang, Shuai Gao, Susan Patalano, Jill A. Macoska, Kourosh Zarringhalam, Dong Han, Changmeng Cai
Materiálatiipa: Artihkal
Giella:English
Almmustuhtton: Frontiers Media S.A. 2022-11-01
Ráidu:Frontiers in Oncology
Fáttát:
androgen
androgen receptor
AR
prostate cancer
androgen-deprivation therapy
CRPC
Liŋkkat:https://www.frontiersin.org/articles/10.3389/fonc.2022.1021845/full

Interneahtta

https://www.frontiersin.org/articles/10.3389/fonc.2022.1021845/full

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