MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface

The type one interferon induced restriction factor Myxovirus resistance B (MxB) restricts HIV-1 nuclear entry evidenced by inhibition of 2-LTR but not linear forms of viral DNA. The HIV-1 capsid is the key determinant of MxB sensitivity and cofactor binding defective HIV-1 capsid mutants P90A (defec...

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Main Authors: Richard J Miles, Claire Kerridge, Laura Hilditch, Christopher Monit, David A Jacques, Greg J Towers
Format: Article
Language:English
Published: eLife Sciences Publications Ltd 2020-06-01
Series:eLife
Subjects:
Online Access:https://elifesciences.org/articles/56910
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author Richard J Miles
Claire Kerridge
Laura Hilditch
Christopher Monit
David A Jacques
Greg J Towers
author_facet Richard J Miles
Claire Kerridge
Laura Hilditch
Christopher Monit
David A Jacques
Greg J Towers
author_sort Richard J Miles
collection DOAJ
description The type one interferon induced restriction factor Myxovirus resistance B (MxB) restricts HIV-1 nuclear entry evidenced by inhibition of 2-LTR but not linear forms of viral DNA. The HIV-1 capsid is the key determinant of MxB sensitivity and cofactor binding defective HIV-1 capsid mutants P90A (defective for cyclophilin A and Nup358 recruitment) and N74D (defective for CPSF6 recruitment) have reduced dependency on nuclear transport associated cofactors, altered integration targeting preferences and are not restricted by MxB expression. This has suggested that nuclear import mechanism may determine MxB sensitivity. Here we have use genetics to separate HIV-1 nuclear import cofactor dependence from MxB sensitivity. We provide evidence that MxB sensitivity depends on HIV-1 capsid conformation, rather than cofactor recruitment. We show that depleting CPSF6 to change nuclear import pathway does not impact MxB sensitivity, but mutants that recapitulate the effect of Cyclophilin A binding on capsid conformation and dynamics strongly impact MxB sensitivity. We demonstrate that HIV-1 primary isolates have different MxB sensitivities due to cytotoxic T lymphocyte (CTL) selected differences in Gag sequence but similar cofactor dependencies. Overall our work demonstrates a complex relationship between cyclophilin dependence and MxB sensitivity likely driven by CTL escape. We propose that cyclophilin binding provides conformational flexibility to HIV-1 capsid facilitating simultaneous evasion of capsid-targeting restriction factors including TRIM5 as well as MxB.
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spelling doaj.art-63d4946b0ef442e1ae53f1f0260db6122022-12-22T02:04:58ZengeLife Sciences Publications LtdeLife2050-084X2020-06-01910.7554/eLife.56910MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surfaceRichard J Miles0Claire Kerridge1Laura Hilditch2Christopher Monit3David A Jacques4https://orcid.org/0000-0002-6426-4510Greg J Towers5https://orcid.org/0000-0002-7707-0264Division of Infection and Immunity, University College London, London, United KingdomDivision of Infection and Immunity, University College London, London, United KingdomDivision of Infection and Immunity, University College London, London, United KingdomDivision of Infection and Immunity, University College London, London, United KingdomEMBL Australia Node in Single Molecule Science and ARC Centre of Excellence in Advanced Molecular Imaging, School of Medical Sciences, University of New South Wales, Sydney, AustraliaDivision of Infection and Immunity, University College London, London, United KingdomThe type one interferon induced restriction factor Myxovirus resistance B (MxB) restricts HIV-1 nuclear entry evidenced by inhibition of 2-LTR but not linear forms of viral DNA. The HIV-1 capsid is the key determinant of MxB sensitivity and cofactor binding defective HIV-1 capsid mutants P90A (defective for cyclophilin A and Nup358 recruitment) and N74D (defective for CPSF6 recruitment) have reduced dependency on nuclear transport associated cofactors, altered integration targeting preferences and are not restricted by MxB expression. This has suggested that nuclear import mechanism may determine MxB sensitivity. Here we have use genetics to separate HIV-1 nuclear import cofactor dependence from MxB sensitivity. We provide evidence that MxB sensitivity depends on HIV-1 capsid conformation, rather than cofactor recruitment. We show that depleting CPSF6 to change nuclear import pathway does not impact MxB sensitivity, but mutants that recapitulate the effect of Cyclophilin A binding on capsid conformation and dynamics strongly impact MxB sensitivity. We demonstrate that HIV-1 primary isolates have different MxB sensitivities due to cytotoxic T lymphocyte (CTL) selected differences in Gag sequence but similar cofactor dependencies. Overall our work demonstrates a complex relationship between cyclophilin dependence and MxB sensitivity likely driven by CTL escape. We propose that cyclophilin binding provides conformational flexibility to HIV-1 capsid facilitating simultaneous evasion of capsid-targeting restriction factors including TRIM5 as well as MxB.https://elifesciences.org/articles/56910HIVcapsidcyclophilin Arestriction factorMxB
spellingShingle Richard J Miles
Claire Kerridge
Laura Hilditch
Christopher Monit
David A Jacques
Greg J Towers
MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
eLife
HIV
capsid
cyclophilin A
restriction factor
MxB
title MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
title_full MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
title_fullStr MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
title_full_unstemmed MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
title_short MxB sensitivity of HIV-1 is determined by a highly variable and dynamic capsid surface
title_sort mxb sensitivity of hiv 1 is determined by a highly variable and dynamic capsid surface
topic HIV
capsid
cyclophilin A
restriction factor
MxB
url https://elifesciences.org/articles/56910
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