Voluntary saccade inhibition deficits correlate with extended white-matter cortico-basal atrophy in Huntington's disease

The ability to inhibit automatic versus voluntary saccade commands in demanding situations can be impaired in neurodegenerative diseases such as Huntington's disease (HD). These deficits could result from disruptions in the interaction between basal ganglia and the saccade control system. To in...

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Bibliographic Details
Main Authors: Israel Vaca-Palomares, Brian C. Coe, Donald C. Brien, Aurelio Campos-Romo, Douglas P. Munoz, Juan Fernandez-Ruiz
Format: Article
Language:English
Published: Elsevier 2017-01-01
Series:NeuroImage: Clinical
Online Access:http://www.sciencedirect.com/science/article/pii/S2213158217301389