LCAT facilitates transacylation of 17β-estradiol in the presence of HDL3 subfraction

It has been shown that estrogens need to be metabolized to their hydrophobic estrogen ester derivatives to act as antioxidants in lipoproteins. Data suggest that 17β-estradiol (E2) becomes esterified in LCAT-induced reactions and the esters are transported from HDL particles to LDL and VLDL particle...

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Bibliographic Details
Main Authors: Anna Höckerstedt, Matti J. Tikkanen, Matti Jauhiainen
Format: Article
Language:English
Published: Elsevier 2002-03-01
Series:Journal of Lipid Research
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S0022227520301450