Chronic aryl hydrocarbon receptor activity impairs muscle mitochondrial function with tobacco smoking

Chronic aryl hydrocarbon receptor activity impairs muscle mitochondrial function with tobacco smoking

Abstract Background Accumulating evidence has demonstrated that chronic tobacco smoking directly contributes to skeletal muscle dysfunction independent of its pathological impact to the cardiorespiratory systems. The mechanisms underlying tobacco smoke toxicity in skeletal muscle are not fully resol...

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Bibliographic Details
Main Authors: Liam F. Fitzgerald, Jacob Lackey, Ahmad Moussa, Sohan V. Shah, Ana Maria Castellanos, Shawn Khan, Martin Schonk, Trace Thome, Zachary R. Salyers, Nishka Jakkidi, Kyoungrae Kim, Qingping Yang, Russell T. Hepple, Terence E. Ryan
Format: Article
Language:English
Published: Wiley 2024-04-01
Series:Journal of Cachexia, Sarcopenia and Muscle
Subjects:
Atrophy
Cigarette
Dioxin
Skeletal muscle
Online Access:https://doi.org/10.1002/jcsm.13439

Internet

https://doi.org/10.1002/jcsm.13439

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