MiR-22 may Suppress Fibrogenesis by Targeting TGFβR I in Cardiac Fibroblasts

MiR-22 may Suppress Fibrogenesis by Targeting TGFβR I in Cardiac Fibroblasts

Background/Aims: Cardiac fibrosis after myocardial infarction (MI) has been identified as a key factor in the development of heart failure, but the mechanisms undelying cardiac fibrosis remained unknown. microRNAs (miRNAs) are novel mechanisms leading to fibrotic diseases, including cardiac fibrosis...

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Bibliographic Details
Main Authors: Yuan Hong, Huaming Cao, Qiang Wang, Jianlin Ye, Lijun Sui, Jinhua Feng, Xiaojun Cai, Huizhu Song, Xiuhong Zhang, Xichuang Chen
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2016-12-01
Series:Cellular Physiology and Biochemistry
Subjects:
Myocardial infarction
Cardiac fibrosis
MicroRNA-22
TGFβRI
Online Access:http://www.karger.com/Article/FullText/453187

Internet

http://www.karger.com/Article/FullText/453187

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