Hyperglycemia attenuates fibroblast contractility via suppression of TβRII receptor modulated α-smooth muscle actin expression

Hyperglycemia attenuates fibroblast contractility via suppression of TβRII receptor modulated α-smooth muscle actin expression

AbstractNonhealing wounds are a common complication in patients suffering from diabetes with hyperglycemia being the most deteriorating factor for this serious pathological condition. Despite the great body of data, the molecular mechanisms by which high glucose affects cellular physiology are still...

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Bibliographic Details
Main Authors: Alexandar Evangelatov, Georgi Georgiev, Borislav Arabadjiev, Stefan Pankov, Plamen Krastev, Albena Momchilova, Roumen Pankov
Format: Article
Language:English
Published: Taylor & Francis Group 2022-12-01
Series:Biotechnology & Biotechnological Equipment
Subjects:
hyperglycemia
cell contractility
α-smooth muscle actin
fibroblast
TGF-β receptor
Online Access:https://www.tandfonline.com/doi/10.1080/13102818.2022.2041486

Internet

https://www.tandfonline.com/doi/10.1080/13102818.2022.2041486

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