Early mitochondrial dysfunction leads to altered redox chemistry underlying pathogenesis of TPI deficiency

Early mitochondrial dysfunction leads to altered redox chemistry underlying pathogenesis of TPI deficiency

Triose phosphate isomerase (TPI) is responsible for the interconversion of dihydroxyacetone phosphate to glyceraldehyde-3-phosphate in glycolysis. Point mutations in this gene are associated with a glycolytic enzymopathy called TPI deficiency. This study utilizes a Drosophila melanogaster model of T...

書誌詳細
主要な著者:	Stacy L. Hrizo, Isaac J. Fisher, Daniel R. Long, Joshua A. Hutton, Zhaohui Liu, Michael J. Palladino
フォーマット:	論文
言語:	English
出版事項:	Elsevier 2013-06-01
シリーズ:	Neurobiology of Disease
主題:	Oxidative stress Redox TPI deficiency Triose phosphate isomerase Glycolytic enzymopathy Drosophila
オンライン･アクセス:	http://www.sciencedirect.com/science/article/pii/S0969996113000119

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