Early mitochondrial dysfunction leads to altered redox chemistry underlying pathogenesis of TPI deficiency

Early mitochondrial dysfunction leads to altered redox chemistry underlying pathogenesis of TPI deficiency

Triose phosphate isomerase (TPI) is responsible for the interconversion of dihydroxyacetone phosphate to glyceraldehyde-3-phosphate in glycolysis. Point mutations in this gene are associated with a glycolytic enzymopathy called TPI deficiency. This study utilizes a Drosophila melanogaster model of T...

Detaylı Bibliyografya
Asıl Yazarlar:	Stacy L. Hrizo, Isaac J. Fisher, Daniel R. Long, Joshua A. Hutton, Zhaohui Liu, Michael J. Palladino
Materyal Türü:	Makale
Dil:	English
Baskı/Yayın Bilgisi:	Elsevier 2013-06-01
Seri Bilgileri:	Neurobiology of Disease
Konular:	Oxidative stress Redox TPI deficiency Triose phosphate isomerase Glycolytic enzymopathy Drosophila
Online Erişim:	http://www.sciencedirect.com/science/article/pii/S0969996113000119

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