Oyster-Derived Tyr-Ala (YA) Peptide Prevents Lipopolysaccharide/D-Galactosamine-Induced Acute Liver Failure by Suppressing Inflammatory, Apoptotic, Ferroptotic, and Pyroptotic Signals

Oyster-Derived Tyr-Ala (YA) Peptide Prevents Lipopolysaccharide/D-Galactosamine-Induced Acute Liver Failure by Suppressing Inflammatory, Apoptotic, Ferroptotic, and Pyroptotic Signals

Models created by the intraperitoneal injection of lipopolysaccharide (LPS) and D-galactosamine (D-GalN) have been widely used to study the pathogenesis of human acute liver failure (ALF) and drug development. Our previous study reported that oyster (Crassostrea gigas) hydrolysate (OH) had a hepatop...

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Bibliographic Details
Main Authors: Adrian S. Siregar, Marie Merci Nyiramana, Eun-Jin Kim, Soo Buem Cho, Min Seok Woo, Dong Kun Lee, Seong-Geun Hong, Jaehee Han, Sang Soo Kang, Deok Ryong Kim, Yeung Joon Choi, Dawon Kang
Format: Article
Language:English
Published: MDPI AG 2021-10-01
Series:Marine Drugs
Subjects:
acute liver injury
apoptosis
ferroptosis
inflammation
oyster
peptide
Online Access:https://www.mdpi.com/1660-3397/19/11/614

Internet

https://www.mdpi.com/1660-3397/19/11/614

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