β-arrestin2 functions as a key regulator in the sympathetic-triggered immunodepression after stroke

β-arrestin2 functions as a key regulator in the sympathetic-triggered immunodepression after stroke

Abstract Background Stroke-induced immunodeficiency syndrome (SIDS) is regarded as a protective mechanism for secondary inflammatory injury as well as a contributor to infection complications. Although stroke-induced hyperactivation of the sympathetic system is proved to facilitate SIDS, the involve...

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Bibliographic Details
Main Authors: Huan Wang, Qi-Wen Deng, Ai-Ni Peng, Fang-Lan Xing, Lei Zuo, Shuo Li, Zheng-Tian Gu, Fu-Ling Yan
Format: Article
Language:English
Published: BMC 2018-04-01
Series:Journal of Neuroinflammation
Subjects:
Stroke
Sympathetic pathway
Immunodepression
β-arrestin2
NF-κB
Online Access:http://link.springer.com/article/10.1186/s12974-018-1142-4

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http://link.springer.com/article/10.1186/s12974-018-1142-4

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