Atypical cell death and insufficient matrix organization in long-bone growth plates from Tric-b-knockout mice

Atypical cell death and insufficient matrix organization in long-bone growth plates from Tric-b-knockout mice

Abstract TRIC-A and TRIC-B proteins form homotrimeric cation-permeable channels in the endoplasmic reticulum (ER) and nuclear membranes and are thought to contribute to counterionic flux coupled with store Ca2+ release in various cell types. Serious mutations in the TRIC-B (also referred to as TMEM3...

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Bibliographic Details
Main Authors: Atsuhiko Ichimura, Yuu Miyazaki, Hiroki Nagatomo, Takaaki Kawabe, Nobuhisa Nakajima, Ga Eun Kim, Masato Tomizawa, Naoki Okamoto, Shinji Komazaki, Sho Kakizawa, Miyuki Nishi, Hiroshi Takeshima
Format: Article
Language:English
Published: Nature Publishing Group 2023-12-01
Series:Cell Death and Disease
Online Access:https://doi.org/10.1038/s41419-023-06285-y

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https://doi.org/10.1038/s41419-023-06285-y

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