Crosstalk between JNK and SUMO signaling pathways: deSUMOylation is protective against H2O2-induced cell injury.

Crosstalk between JNK and SUMO signaling pathways: deSUMOylation is protective against H2O2-induced cell injury.

Oxidative stress is a key feature in the pathogenesis of several neurological disorders. Following oxidative stress stimuli a wide range of pathways are activated and contribute to cellular death. The mechanism that couples c-Jun N-terminal kinase (JNK) signaling, a key pathway in stress conditions,...

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Bibliographic Details
Main Authors: Marco Feligioni, Elisa Brambilla, Agata Camassa, Alessandra Sclip, Andrea Arnaboldi, Federica Morelli, Xanthi Antoniou, Tiziana Borsello
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2011-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3229511?pdf=render

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http://europepmc.org/articles/PMC3229511?pdf=render

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