TRPC4 deletion elicits behavioral defects in sociability by dysregulating expression of microRNA-138-2
Summary: To investigate whether the defects in transient receptor potential canonical 4 (TRPC4), which is strongly expressed in the hippocampus, are implicated in ASD, we examined the social behaviors of mice in which Trpc4 was deleted (Trpc4−/−). Trpc4−/− mice displayed the core symptoms of ASD, na...
| Үндсэн зохиолчид: | , , , , , , , , |
|---|---|
| Формат: | Өгүүллэг |
| Хэл сонгох: | English |
| Хэвлэсэн: |
Elsevier
2024-01-01
|
| Цуврал: | iScience |
| Нөхцлүүд: | |
| Онлайн хандалт: | http://www.sciencedirect.com/science/article/pii/S2589004223026949 |