FAK mediates hypoxia‐induced pulmonary artery smooth muscle cell proliferation by modulating mitochondrial transcription termination factor 1/cyclin D1

Abstract This study aimed to investigate the mechanism of FAK‐dependent hypoxia‐induced proliferation on human pulmonary artery smooth muscle cells (HPASMCs). Primary HPASMCs were isolated and cultured in vitro under normal and hypoxia conditions to assess cell proliferation with cell counting kit‐8...

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Bibliographic Details
Main Authors: Chunlong Lin, Hui Yang, Qiong Luo, Qi Liu
Format: Article
Language:English
Published: Wiley 2024-03-01
Series:Clinical and Translational Science
Online Access:https://doi.org/10.1111/cts.13767