USP13 ameliorates nonalcoholic fatty liver disease through inhibiting the activation of TAK1

USP13 ameliorates nonalcoholic fatty liver disease through inhibiting the activation of TAK1

Abstract Background The molecular mechanisms underlying nonalcoholic fatty liver disease (NAFLD) remain to be fully elucidated. Ubiquitin specific protease 13 (USP13) is a critical participant in inflammation-related signaling pathways, which are linked to NAFLD. Herein, the roles of USP13 in NAFLD...

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Bibliographic Details
Main Authors: Min Tang, Han Cao, Yunqin Ma, Shuangshuang Yao, Xiaohui Wei, Yijiong Tan, Fang liu, Yongde Peng, Nengguang Fan
Format: Article
Language:English
Published: BMC 2024-07-01
Series:Journal of Translational Medicine
Subjects:
Ubiquitin specific protease 13
Transforming growth factor beta-activated Kinase 1
Nonalcoholic fatty liver disease
Ubiquitination
Online Access:https://doi.org/10.1186/s12967-024-05465-4

Internet

https://doi.org/10.1186/s12967-024-05465-4

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