Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain
Acyl CoA:monoacylglycerol acyltransferase 2 (MGAT2) is thought to be crucial for dietary fat absorption. Indeed, mice lacking the enzyme (Mogat2−/−) are resistant to obesity and other metabolic disorders induced by high-fat feeding. However, these mice absorb normal quantities of fat. To explore whe...
| Main Authors: | , , , , |
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| 格式: | 文件 |
| 语言: | English |
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Elsevier
2011-09-01
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| 丛编: | Journal of Lipid Research |
| 主题: | |
| 在线阅读: | http://www.sciencedirect.com/science/article/pii/S0022227520408636 |