Thiazolidinediones promote axonal growth through the activation of the JNK pathway.
The axon is a neuronal process involved in protein transport, synaptic plasticity, and neural regeneration. It has been suggested that their structure and function are profoundly impaired in neurodegenerative diseases. Previous evidence suggest that Peroxisome Proliferator-Activated Receptors-γ (PPA...
Main Authors: | Rodrigo A Quintanilla, Juan A Godoy, Ivan Alfaro, Deny Cabezas, Rommy von Bernhardi, Miguel Bronfman, Nibaldo C Inestrosa |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2013-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC3669289?pdf=render |
Similar Items
-
Phosphorylated tau potentiates Aβ-induced mitochondrial damage in mature neurons
by: Rodrigo A. Quintanilla, et al.
Published: (2014-11-01) -
Acetylcholinesterase induces the expression of the β-amyloid precursor protein in glia and activates glial cells in culture
by: Rommy von Bernhardi, et al.
Published: (2003-12-01) -
How the Wnt signaling pathway protects from neurodegeneration: The Mitochondrial Scenario
by: Macarena S. Arrázola, et al.
Published: (2015-05-01) -
El Desafío de envejecer: Una mirada desde la neurociencia
by: Rommy von Bernhardi
Published: (2019-03-01) -
Age-dependent changes on fractalkine forms and their contribution to neurodegenerative diseases
by: Jaime Eugenín, et al.
Published: (2023-09-01)