Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na<sub>V</sub>1.7 produce distinct pain disorders
<p>Abstract</p> <p>Background</p> <p>Two groups of gain-of-function mutations in sodium channel Na<sub>V</sub>1.7, which are expressed in dorsal root ganglion (DRG) neurons, produce two clinically-distinct pain syndromes - inherited erythromelalgia (IEM) and...
| Main Authors: | , , , , , |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
SAGE Publishing
2010-04-01
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| Series: | Molecular Pain |
| Online Access: | http://www.molecularpain.com/content/6/1/24 |