MiR 208a Regulates Mitochondrial Biogenesis in Metabolically Challenged Cardiomyocytes

MiR 208a Regulates Mitochondrial Biogenesis in Metabolically Challenged Cardiomyocytes

Metabolic syndrome increases the risk for cardiovascular disease including metabolic cardiomyopathy that may progress to heart failure. The decline in mitochondrial metabolism is considered a critical pathogenic mechanism that drives this progression. Considering its cardiac specificity, we hypothes...

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Bibliographic Details
Main Authors: Naveen Mekala, Jacob Kurdys, Alexis Paige Vicenzi, Leana Rose Weiler, Carmen Avramut, Edwin J. Vazquez, Neli Ragina, Mariana G. Rosca
Format: Article
Language:English
Published: MDPI AG 2021-11-01
Series:Cells
Subjects:
cardiomyocytes
metabolic syndrome
miR 208a
mitochondrial biogenesis
bioenergetics
Online Access:https://www.mdpi.com/2073-4409/10/11/3152

Internet

https://www.mdpi.com/2073-4409/10/11/3152

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