Acute BDNF treatment upregulates GluR1-SAP97 and GluR2-GRIP1 interactions: implications for sustained AMPA receptor expression.
Brain-derived neurotrophic factor (BDNF) plays several prominent roles in synaptic plasticity and in learning and memory formation. Reduced BDNF levels and altered BDNF signaling have been reported in several brain diseases and behavioral disorders, which also exhibit reduced levels of AMPAr subunit...
| Հիմնական հեղինակներ: | , |
|---|---|
| Ձևաչափ: | Հոդված |
| Լեզու: | English |
| Հրապարակվել է: |
Public Library of Science (PLoS)
2013-01-01
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| Շարք: | PLoS ONE |
| Առցանց հասանելիություն: | https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23460828/?tool=EBI |