Endogenous sulfur dioxide deficiency as a driver of cardiomyocyte senescence through abolishing sulphenylation of STAT3 at cysteine 259

Endogenous sulfur dioxide deficiency as a driver of cardiomyocyte senescence through abolishing sulphenylation of STAT3 at cysteine 259

Objective: Cardiomyocyte senescence is an important contributor to cardiovascular diseases and can be induced by stressors including DNA damage, oxidative stress, mitochondrial dysfunction, epigenetic regulation, etc. However, the underlying mechanisms for the development of cardiomyocyte senescence...

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Bibliographic Details
Main Authors: Shangyue Zhang, Bingquan Qiu, Boyang Lv, Guosheng Yang, Yinghong Tao, Yongyan Hu, Kun Li, Xiaoqi Yu, Chaoshu Tang, Junbao Du, Hongfang Jin, Yaqian Huang
Format: Article
Language:English
Published: Elsevier 2024-05-01
Series:Redox Biology
Subjects:
Sulfur dioxide
Cardiomyocyte senescence
DNA damage
STAT3
Sulphenylation
Online Access:http://www.sciencedirect.com/science/article/pii/S2213231724001009

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http://www.sciencedirect.com/science/article/pii/S2213231724001009

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