Prenatal infection predisposes offspring to enhanced susceptibility to imiquimod-mediated psoriasiform dermatitis in mice

Background: Infection can upregulate T helper 17 (Th17) signaling and exacerbate psoriasis. Literature has indicated that prenatal infection induces embryonic development toward Th17 signaling and is a risk factor for developing certain Th17-skewing disorders such as autism in offspring. However, it...

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Bibliographic Details
Main Authors: Sebastian Yu, Chia-Wen Lee, Yu-An Li, Tsung-Hsi Chen, Hsin-Su Yu
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2022-01-01
Series:Dermatologica Sinica
Subjects:
Online Access:http://www.dermsinica.org/article.asp?issn=1027-8117;year=2022;volume=40;issue=1;spage=14;epage=19;aulast=Yu