A highly penetrant form of childhood apraxia of speech due to deletion of 16p11.2

A highly penetrant form of childhood apraxia of speech due to deletion of 16p11.2

Individuals with heterozygous 16p11.2 deletions reportedly suffer from a variety of difficulties with speech and language. Indeed, recent copy-number variant screens of children with childhood apraxia of speech (CAS), a specific and rare motor speech disorder, have identified three unrelated individ...

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Bibliographic Details
Main Authors: Fedorenko, Evelina, Morgan, Angela, Murray, Elizabeth, Mei, Cristina, Tager-Flusberg, Helen, Fisher, Simon E, Cardinaux, Anne, Kanwisher, Nancy
Other Authors: Massachusetts Institute of Technology. Department of Brain and Cognitive Sciences
Format: Article
Language:en_US
Published: Nature Publishing Group 2018
Online Access:http://hdl.handle.net/1721.1/113018
https://orcid.org/0000-0003-3853-7885

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http://hdl.handle.net/1721.1/113018
https://orcid.org/0000-0003-3853-7885

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