Persistent Reversal of Enhanced Amphetamine Intake by Transient CaMKII Inhibition

Persistent Reversal of Enhanced Amphetamine Intake by Transient CaMKII Inhibition

Amphetamine exposure transiently increases Ca2+/calmodulin-dependent protein kinase II (CaMKII) α expression in the nucleus accumbens (NAcc) shell and this persistently increases local GluA1 S831 phosphorylation and enhances behavioral responding to the drug. Here we assessed whether transiently int...

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Bibliographic Details
Main Authors: Loweth, Jessica A., Li, Dongdong, Cortright, James J., Wilke, Georgia, Jeyifous, Okunola, Neve, Rachael L., Bayer, K. Ulrich, Vezina, Paul
Other Authors: Massachusetts Institute of Technology. Department of Brain and Cognitive Sciences
Format: Article
Language:en_US
Published: Society for Neuroscience 2013
Online Access:http://hdl.handle.net/1721.1/79759
https://orcid.org/0000-0002-3854-5968

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http://hdl.handle.net/1721.1/79759
https://orcid.org/0000-0002-3854-5968

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