Review on the functions of Rasd1 interacting proteins.

Smith-Magenis syndrome (SMS) is a genetic disease associated with an interstitial deletion of band p11.2 on chromosome 17. Rasd1 is one of the genes in the minimum deletion interval necessary to produce the SMS phenotype. Besides being a non-receptor activator of G-protein signaling in mammalian cel...

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Bibliographic Details
Main Author: Liew, Jasmine Jek Peng.
Other Authors: Chen Ken-Shiung
Format: Final Year Project (FYP)
Language:English
Published: 2009
Subjects:
Online Access:http://hdl.handle.net/10356/15456