Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A

Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A

We previously reported that hepatitis C virus (HCV) infection activates the reactive oxygen species (ROS)/c-Jun N-terminal kinase (JNK) signaling pathway. However, the roles of ROS/JNK activation in the HCV life cycle remain unclear. We sought to identify a novel role of the ROS/JNK signaling pathwa...

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Bibliographic Details
Main Authors: Deng, Lin, Liang, Yujiao, Ariffianto, Adi, Matsui, Chieko, Abe, Takayuki, Muramatsu, Masamichi, Wakita, Takaji, Maki, Masatoshi, Shibata, Hideki, Shoji, Ikuo
Format: Article
Language:English
Published: American Society for Microbiology 2022
Subjects:
Medical Biochemistry and Metabolomics
Infectious Diseases
Online Access:https://repository.ugm.ac.id/279044/1/Hepatitis%20C%20Virus-Induced%20ROS%20%20JNK%20Signaling%20Pathway%20Activates%20the%20E3%20Ubiquitin%20Ligase%20Itch%20to%20Promote%20the%20Release%20of%20HCV%20Particles%20via%20Polyubiquitylation%20of%20VPS4A.pdf

Internet

https://repository.ugm.ac.id/279044/1/Hepatitis%20C%20Virus-Induced%20ROS%20%20JNK%20Signaling%20Pathway%20Activates%20the%20E3%20Ubiquitin%20Ligase%20Itch%20to%20Promote%20the%20Release%20of%20HCV%20Particles%20via%20Polyubiquitylation%20of%20VPS4A.pdf

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