GLUCOSE DOES NOT BLOCK ATP-REGULATED K+-CHANNEL ACTIVITY IN FETAL B-CELLS

GLUCOSE DOES NOT BLOCK ATP-REGULATED K+-CHANNEL ACTIVITY IN FETAL B-CELLS

Bibliografische gegevens
Hoofdauteurs:	Welsh, M, Berggren, P, Rorsman, P
Formaat:	Journal article
Gepubliceerd in:	1987

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Glucose dependent K+-channels in pancreatic beta-cells are regulated by intracellular ATP.
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Mechanism of K-ATP channel inhibition by ATP
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Differential metabolic and nucleotide sensitivity of beta-cell and cardiac K-ATP channels
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Opposite effects of tolbutamide and diazoxide on the ATP-dependent K+ channel in mouse pancreatic beta-cells.
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How ATP inhibits the open K(ATP) channel.
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Sulfonylurea-mediated stimulation of insulin exocytosis via an ATP-sensitive K+ channel-independent action.
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ATP-regulated potassium channels and voltage-gated calcium channels in pancreatic alpha and beta cells: Similar functions but reciprocal effects on secretion
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Molecular mechanism of sulphonylurea block of K(ATP) channels carrying mutations that impair ATP inhibition and cause neonatal diabetes.
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Activation by adrenaline of a low-conductance G protein-dependent K+ channel in mouse pancreatic B cells.
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Glucose-inhibition of glucagon secretion involves activation of GABAA-receptor chloride channels.
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Dynamic sensitivity of K-ATP channels to ATP
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ADP MODULATES THE ATP-SENSITIVITY OF THE K-CHANNEL CLOSED BY GLUCOSE IN ISOLATED RAT PANCREATIC B-CELLS
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