Investigation into the onset and progression of transplant arteriosclerosis in a mice aortic retransplantation model.

Investigation into the onset and progression of transplant arteriosclerosis in a mice aortic retransplantation model.

Long-term function of vascularized human organ grafts is often limited by transplant arteriosclerosis and can lead to graft failure. Here, we have analyzed the impact of an initial rejection episode on the later development of transplant arteriosclerosis. Following transplantation of allogeneic abdo...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Steger, U, Ensminger, S, Bushell, A, Wood, K
Μορφή:	Journal article
Γλώσσα:	English
Έκδοση:	2008

Παρόμοια τεκμήρια

Development of a combined cardiac and aortic transplant model to investigate the development of transplant arteriosclerosis in the mouse.
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Comparison of the effects of exposure to a single or multiple donor alloantigens in the development of transplant arteriosclerosis.
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Neutralizing interleukin-4 prevents transplant arteriosclerosis mediated by indirect pathway T cells under CD40-CD154 costimulation blockade.
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The persistence of transplant arteriosclerosis despite CD154 blockade.
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Platelet-endothelial cell adhesion molecule-1 (CD31) expression on donor endothelial cells attenuates the development of transplant arteriosclerosis.
ανά: Ensminger, S, κ.ά.
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Kinetics of transplant arteriosclerosis in MHC-Class I mismatched and fully allogeneic mouse aortic allografts.
ανά: Ensminger, S, κ.ά.
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Critical role for IL-4 in the development of transplant arteriosclerosis in the absence of CD40-CD154 costimulation.
ανά: Ensminger, S, κ.ά.
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Indirect allorecognition plays a pivotal role in the pathogenesis of transplant arteriosclerosis.
ανά: Ensminger, S, κ.ά.
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Regulation of transplant arteriosclerosis by CD25+CD4+ T cells generated to alloantigen in vivo.
ανά: Warnecke, G, κ.ά.
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Indirect allorecognition can play an important role in the development of transplant arteriosclerosis.
ανά: Ensminger, S, κ.ά.
Έκδοση: (2002)

CD8+ T cells contribute to the development of transplant arteriosclerosis despite CD154 blockade.
ανά: Ensminger, S, κ.ά.
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Tadalafil attenuates graft arteriosclerosis of aortic transplant in a rat model
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Mouse endothelial CD40 expression does not play a role during the development of transplant arteriosclerosis.
ανά: Ensminger, S, κ.ά.
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Eosinophils are major effector cells for the development of transplant arteriosclerosis and depend on the presence of IL-4.
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Increased transplant arteriosclerosis in the absence of CCR7 is associated with reduced expression of Foxp3.
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CD4+ regulatory T cells generated in vitro with IFN-{gamma} and allogeneic APC inhibit transplant arteriosclerosis.
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In vivo prevention of transplant arteriosclerosis by ex vivo-expanded human regulatory T cells.
ανά: Nadig, SN, κ.ά.
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Intragraft interleukin-4 mRNA expression after short-term CD154 blockade may trigger delayed development of transplant arteriosclerosis in the absence of CD8+ T cells.
ανά: Ensminger, S, κ.ά.
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Immune-Mediated Vascular Injury and Dysfunction in Transplant Arteriosclerosis
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Ex vivo expanded human treg inhibit the development of transplant arteriosclerosis in a humanized in vivo model
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Rehabilitative goals for patients undergoing lung retransplantation
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Response to "reconstruction of the hepatic artery in liver transplantation in mice: Mandatory or useless?"
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Low-dose rapamycin treatment increases the ability of human regulatory T cells to inhibit transplant arteriosclerosis in vivo.
ανά: Hester, J, κ.ά.
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Low-dose rapamycin treatment increases the ability of human regulatory T cells to inhibit transplant arteriosclerosis in vivo.
ανά: Hester, J, κ.ά.
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Complex arterial reconstruction for pancreas transplantation in recipients with advanced arteriosclerosis.
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Early arteriosclerosis and its risk factors in subjects with prediabetes and new-onset diabetes
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