HCM and DCM mutations in thin filament proteins have opposite effects on regulation of contractility in vitro
Autors principals: | Redwood, C, Robinson, P, Mirza, M, Knott, A, Marston, S, Watkins, H |
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Format: | Conference item |
Publicat: |
2003
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Ítems similars
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HCM and DCM mutations in cardiac thin filament proteins have opposite effects on the regulation of contractility in vitro
per: Redwood, C, et al.
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HCM and DCM mutations in thin filament proteins have opposite effects on in vitro motility
per: Marston, S, et al.
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Effects of DCM mutations in thin filament regulatory proteins on contractile function
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Mutations in tropomyosin that cause DCM, affect cooperativity of cardiac muscle thin filaments
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