Increased GTP-cyclohydrolase I expression but not vitamin C treatment restored accelerated atherosclerotic lesion formation in apolipoprotein E-deficient mice overexpressing endothelial nitric oxide synthase

Παρόμοια τεκμήρια

• GTPCH I overexpression decreases atherosclerotic lesion formation in apolipoprotein E-deficient/eNOS transgenic mice
  ανά: Takaya, T, κ.ά.
  Έκδοση: (2004)
• Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
  ανά: Bendall, J, κ.ά.
  Έκδοση: (2004)
• Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
  ανά: Alp, N, κ.ά.
  Έκδοση: (2003)
• Endothelial-specific GTP cyclohydrolase overexpression reduces blood pressure in salt-sensitive hypertension
  ανά: Chen, A, κ.ά.
  Έκδοση: (2006)
• Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
  ανά: Bendall, J, κ.ά.
  Έκδοση: (2005)