Tetrahydrobiopterin (BH4) is essential for NO-mediated flow-dependent dilation of isolated skeletal muscle arterioles

Tetrahydrobiopterin (BH4) is essential for NO-mediated flow-dependent dilation of isolated skeletal muscle arterioles

Podrobná bibliografie
Hlavní autoři:	Bagi, Z, Koller, A
Médium:	Conference item
Vydáno:	2003

Podobné jednotky

Chronic renal failure leads to reduced flow-dependent dilation in isolated rat skeletal muscle arterioles due to lack of NO mediation.
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Lack of flow-mediated dilation and enhanced angiotensin II-induced constriction in skeletal muscle arterioles of lupus-prone autoimmune mice.
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Impaired NO-mediation of flow dependent arteriolar dilation in diabetes due to reduced level of tetrahydrobiopterin
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Transient elevation of glucose concentration reduces NO-mediation of flow-induced arteriolar dilation due to interaction between superoxide and tetrahydrobiopterin (BH4)
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Nitric oxide and hydrogen peroxide contribute to the development of reactive dilation in isolated coronary arterioles
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PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress.
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Nitric oxide and H2O2 contribute to reactive dilation of isolated coronary arterioles.
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Correction: Voltage-dependent inward currents in smooth muscle cells of skeletal muscle arterioles.
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Enhanced superoxide production reduces NO-mediation of flow-dependent dilation of isolated coronary arteries in type 2 diabetes mellitus
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Flow-induced constriction in arterioles of hyperhomocysteinemic rats is due to impaired nitric oxide and enhanced thromboxane A(2) mediation.
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Hydrogen peroxide elicits constriction of skeletal muscle arterioles by activating the arachidonic acid pathway.
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Tetrahydrobiopterin (Bh4) deficiencies: diagnosis and treatment
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Lack of nitric oxide mediation of flow-dependent arteriolar dilation in type I diabetes is restored by sepiapterin.
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High intraluminal pressure reduces tachyphylaxis to angiotensin II in isolated arterioles
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Prenatal Diagnosis of Tetrahydrobiopterin (BH4) Synthase Deficiency
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Simultaneously increased TxA(2) activity in isolated arterioles and platelets of rats with hyperhomocysteinemia.
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Consensus guideline for the diagnosis and treatment of tetrahydrobiopterin (BH4) deficiencies
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Endothelium-dependent vasodilation in myogenically active mouse skeletal muscle arterioles: role of EDH and K(+) channels.
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Multiple effects of diabetes mellitus on the vasomotor responses of human coronary arterioles
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20-hydroxyecdysone dilates muscle arterioles in a nitric oxide-dependent, estrogen ER-β receptor-independent manner
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Hydrogen peroxide elicits COX-2-dependent production of constrictor prostaglandins in arterioles of type 2 diabetic mice
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Correction to: Consensus guideline for the diagnosis and treatment of tetrahydrobiopterin (BH4) deficiencies
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PPARgamma activation, by reducing oxidative stress, increases NO bioavailability in coronary arterioles of mice with Type 2 diabetes.
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P683The role of nitric oxide synthase (NOS) and its essential cofactor tetrahydrobiopterin (BH4) in diabetic cardiomyopathy.
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