Inhibition of hemangiogenesis and lymphangiogenesis after normal-risk corneal transplantation by neutralizing VEGF promotes graft survival

Inhibition of hemangiogenesis and lymphangiogenesis after normal-risk corneal transplantation by neutralizing VEGF promotes graft survival

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Xehetasun bibliografikoak
Egile Nagusiak:	Cursiefen, C, Chen, L, Maruyama, K, Jackson, D, Dana, R, Wiegand, S, Streilein, J
Formatua:	Conference item
Argitaratua:	2004

Antzeko izenburuak

Inhibition of hemangiogenesis and lymphangiogenesis after normal-risk corneal transplantation by neutralizing VEGF promotes graft survival.
nork: Cursiefen, C, et al.
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VEGF-A stimulates lymphangiogenesis and hemangiogenesis in inflammatory neovascularization via macrophage recruitment.
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Complete inhibition of simultaneous onset corneal lymphangiogenesis and angiogenesis by trapping vascular endothelial growth factor (VEGF) using a VEGF receptor 1 and 2 chimeric protein (VEGF TrapR1R2)
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Time course of angiogenesis and lymphangiogenesis after brief corneal inflammation.
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Spontaneous corneal hem- and lymphangiogenesis in mice with destrin-mutation depend on VEGFR3 signaling.
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Inflammation-induced lymphangiogenesis in the cornea arises from CD11b-positive macrophages.
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Induction of tumor lymphangiogenesis by VEGF-C promotes breast cancer metastasis.
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Hyaluronan derived from the limbus is a key Regulator of Corneal Lymphangiogenesis
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Hyaluronan derived from the limbus is a key regulator of corneal lymphangiogenesis
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Hyaluronan derived from the limbus is a key regulator of corneal lymphangiogenesis
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VEGF-C gene therapy augments postnatal lymphangiogenesis and ameliorates secondary lymphedema.
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Human mesenchymal stem cells derived from adipose tissue showed a more robust effect than those from the umbilical cord in promoting corneal graft survival by suppressing lymphangiogenesis
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