Cellular response to DNA interstrand crosslinks: The Fanconi Anemia pathway
Interstrand crosslinks (ICLs) are a highly toxic form of DNA damage. ICLs can interfere with vital biological processes requiring separation of the two DNA strands, such as replication and transcription. If ICLs are left unrepaired, it can lead to mutations, chromosome breakage and mitotic catastrop...
المؤلفون الرئيسيون: | Lopez-Martinez, D, Liang, C, Cohn, M |
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التنسيق: | Journal article |
منشور في: |
Springer Verlag
2016
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مواد مشابهة
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UHRF1 Is a sensor for DNA interstrand crosslinks and recruits FANCD2 to initiate the Fanconi Anemia pathway
حسب: Liang, C, وآخرون
منشور في: (2015) -
UHRF1 Is a Sensor for DNA Interstrand Crosslinks and Recruits FANCD2 to Initiate the Fanconi Anemia Pathway
حسب: Chih-Chao Liang, وآخرون
منشور في: (2015-03-01) -
UHRF1 is a sensor for DNA interstrand crosslinks.
حسب: Cohn, M, وآخرون
منشور في: (2016) -
Elevated levels of STAT1 in Fanconi anemia group A lymphoblasts correlate with the cells’ sensitivity to DNA interstrand crosslinking drugs
حسب: Inés Prieto-Remón, وآخرون
منشور في: (2013-05-01) -
Components of a Fanconi-like pathway control Pso2-independent DNA interstrand crosslink repair in yeast
حسب: Ward, T, وآخرون
منشور في: (2012)